This probiotic bacterium was originally isolated from the intestinal tract of humans.
Bacillus bifidus is a specific inhabitant of the large intestine (colon) and composes a large presence of the beneficial microflora which produce acids to retard colonization of putrefactive bacteria. Bacillus bifidus composes a majority of the beneficial microflora which produce acids to retard colonization of putrefactive bacteria such as E. coli, Clostridium, and Salmonella. Strains of this species have been used in the production of foods containing bifidobacteria, such as cultured milks, and in therapeutic preparations for the treatment of the following: digestive disorders in infants, enterocolitis, constipation, cirrhosis of the liver, imbalance of intestinal flora following antibiotic therapy, and for promotion of intestinal peristalsis. •
Bacillus bifidus reduces constipation, diarrhea, colon inflammation and the concentration of dead bacteria buildup; digests lactose & fibers; balances intestinal pH; supports liver; enhances assimilation of minerals; produces B vitamins; inhibits Candida; lowers the ability of E.coli to adhere to intestinal walls.
Bifidobacteria are normal inhabitants of the colon. Newborns, especially those that are breast-fed, are colonized with bifidobacteria within days after birth.
The population of Bifidobacteria in the colon appears to be relatively stable until advanced age when it appears to decline.
The bifidobacteria population is influenced by a number of factors, including diet, antibiotics and stress.
History: Bifidobacteria were first isolated from the feces of breast-fed infants. Bifidobacteria have various shapes, including short, curved rods, club-shaped rods and bifurcated Y-shaped rods. The Bifidobacteria name is derived from the observation that they often exist in a Y-shaped or bifid form.
Technicals: Bifidobacteria are gram-positive anaerobes. Bifidobacteria are non-motile, non-spore forming and catalase-negative.
Bifidobacteria are saccharolytic organisms that produce acetic and lactic acids without generation of CO2, except during degradation of gluconate.
The statements on this Web site have not been evaluated by the Food and Drug Administration (FDA). And are not intended to diagnose, treat, cure or prevent disease. The information presented is not intended to replace medical advice or treatment from your own doctor or healthcare provider. Nothing presented here is intended as a substitute for prescription medication or any other medical treatment prescribed by your doctor.